STRC RBM24 Exon Splicing Quantification

Sun et al. 2026 SD03 dataset (rMATS SE analysis, Rbm24-exon4-KO vs control cochlea) contains 4 significant exon-skipping events in Strc. Two are in-frame and therapeutically relevant.

In-frame events (biologically interpretable)

Eventmm10 coords (chr2 −)SizedPSIFDR
E1121203399–121203567168 nt = 56 aa−0.5422.1 × 10⁻⁵
E2121198816–12119889478 nt = 26 aa−0.4373.7 × 10⁻³

Divisibility test: 168 ÷ 3 = 56.0 ✓ | 78 ÷ 3 = 26.0 ✓

Frameshift events (not interpretable as natural isoforms)

EventSize÷3
E370 nt23.33 ❌
E4197 nt65.67 ❌

Key finding

When RBM24 is absent, STRC exon inclusion drops 44–54% at two in-frame sites. This is consistent with RBM24 acting as a splicing enhancer for these exons — without it, the spliceosome skips them at high frequency.

Resolved (2026-04-20) — human protein mapping

Cross-species aa mapping via Ensembl canonical transcripts + BLOSUM62 alignment. All 4 events fall inside mini-STRC (aa 700–1775). TMEM145 interface empirically extracted from AF3 Job 2 CIF: aa 1603–1770 (C-terminal ARM repeats, not 950–1200 as earlier estimated). Full mapping in STRC RBM24 Exon Mapping to Human Protein.

EventHuman aaIn mini-STRCDistance to TMEM145 interface
E11047–1102501 aa upstream
E21311–1376227 aa upstream
E3712–732✅ (boundary)871 aa upstream
E41311–1376227 aa upstream

None of the events directly overlap the TMEM145 interface. E1 (56 aa) is extremely conserved across mammals (100 % in chimp, 98 % rabbit, 93–95 % rodent/dog/pig), so it is functionally important — but not via TMEM145. E3 and E4 are frameshift events that trigger NMD.

Connections