Calcium balance and mechanotransduction in rat cochlear OHCs quantified by fluorescence imaging and electrophysiology. Provides the primary quantitative source for PMCA Ca2+ extrusion rate and endogenous buffer capacity in the h05 model. The “Mammano 1999 Pflugers Arch” attribution in h05 scripts is wrong; this Beurg 2010 paper is the correct source for k_extrusion.

Key finding

Electrogenic PMCA current saturates at 18 pA → maximum Ca2+ extrusion rate of 3.7 fmol·s⁻¹. Ca2+ clearance time constant after bundle deflection ~50 ms for brief stimuli, 100–300 ms for prolonged. Endogenous mobile buffer equivalent to 1 mM BAPTA (~5 mM Ca2+-binding sites from parvalbumin-β + calbindin-D28k). PMCA density in stereociliary membrane ~6000 pumps/µm².

Numbers that matter

ParameterValueUnitsConditionsNotes
Max PMCA extrusion rate3.7fmol/sRat OHC, saturating Ca2+Electrogenic current 18 pA
Ca2+ clearance τ (brief stimulus)~50msRat OHCFast component; PMCA + diffusion
Ca2+ clearance τ (prolonged)100–300msRat OHCSlower recovery
Endogenous mobile buffer~1mM BAPTA-equivalentOHC apical compartment~5 mM Ca2+-binding sites total
PMCA density (stereocilia)~6000pumps/µm²Rat OHCHigher than bullfrog (Yamoah 1998)
k_extrusion (derived estimate)~30/sSee noteDerived from 3.7 fmol/s ÷ V_apex×[Ca] — model parameter, not directly stated

Derivation note for k_extrusion ~30 /s: At Ca_rest ~30 nM in V_apex = 50 fL, total Ca ~1.5 attomol. Max flux 3.7 fmol/s is 2500× higher than resting pool — so linear first-order approximation k_extrusion30/s is plausible as a near-saturation efficiency parameter, but is not a directly reported number. The paper reports flux not k.

Limitations

  • Rat cochlear OHCs (P10-P14). Postnatal development may affect PMCA density.
  • k_extrusion ~30 /s is a derived model parameter, not a directly reported rate constant from this paper.
  • Buffer capacity measured via BAPTA analog; parvalbumin-β binding kinetics differ from BAPTA (faster on-rate).

Connections